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lifestyle · for kidney stone prevention

Low-Sodium Diet

Reviewed by the Nimbera editorial team · Last updated May 2026

A landmark 5-year RCT (PMID 11784873, NEJM 2002, n=120 men with recurrent calcium oxalate stones and hypercalciuria) found a normal-calcium, low-sodium (50 mmol NaCl/day), low-animal-protein diet reduced stone recurrence risk by 51% compared to a traditional low-calcium diet (RR 0.49, p=0.04).$0/month

How to use

Target under 2,300 mg of sodium per day (about 1 teaspoon of salt total from all sources). Read nutrition labels - processed foods, canned soups, deli meats, and restaurant meals are the largest sources. Cook at home using herbs, lemon, and spices instead of salt. Pair sodium reduction with adequate dietary calcium (1,000-1,200 mg/day from food) to prevent compensatory oxalate absorption from the gut.

How it works

High sodium intake forces the kidneys to excrete more sodium in urine; calcium follows sodium in the proximal tubule, raising urinary calcium concentration and directly increasing the risk of calcium salt crystallization.

Where to buy

No supplement needed - this is a dietary change. Low-sodium food products are widely available at all supermarkets.

Typical cost: $0/month

Safety notes

Do not combine a low-sodium diet with a low-calcium diet - restricting both increases oxalate absorption and can worsen stone risk. Athletes and individuals working in hot climates who lose significant sodium through sweat should not restrict sodium without medical supervision. Some blood pressure medications (e.g. loop diuretics) interact with sodium balance - speak with a doctor before making significant dietary changes if you take medication.

Drug & supplement interactions

  • Always tell your doctor or pharmacist about any supplements you take.
  • Interactions can occur with prescription medications, blood thinners, and immunosuppressants.
  • Herbal remedies can affect how the body absorbs or metabolises drugs.
  • Check drugs.com interactions checker before starting.

Clinical source

NEJM 2002

This information is compiled from primary clinical literature and is for informational purposes only. It is not a substitute for professional medical advice. Read our full disclaimer.